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Semin Respir Crit Care Med 2015; 36(03): 408-421
DOI: 10.1055/s-0035-1554846
Thieme Medical Publishers 333 Seventh Avenue, New York, NY 10001, USA.

Chronic Obstructive Pulmonary Disease Secondary to Household Air Pollution

Nour A. Assad
1   Division of Pulmonary, Critical Care and Sleep Medicine, Department of Internal Medicine, University of New Mexico Health Sciences Center School of Medicine, 1 University of New Mexico, Albuquerque, New Mexico
,
John Balmes
2   Division of Occupational and Environmental Medicine, Department of Internal Medicine, University of California San Francisco School of Medicine, San Francisco, California
3   Division of Environmental Health Sciences, School of Public Health, University of California, Berkeley, California
,
Sumi Mehta
4   Department of Research and Evaluation, Global Alliance For Clean Cookstoves, Pennsylvania, Washington, District of Columbia
,
Umar Cheema
5   Dow University of Health Sciences, Ojha Campus, Karachi, Pakistan
,
Akshay Sood
1   Division of Pulmonary, Critical Care and Sleep Medicine, Department of Internal Medicine, University of New Mexico Health Sciences Center School of Medicine, 1 University of New Mexico, Albuquerque, New Mexico
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Publikationsdatum:
29. Mai 2015 (online)

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Abstract

Approximately 3 billion people around the world cook and heat their homes using solid fuels in open fires and rudimentary stoves, resulting in household air pollution. Household air pollution secondary to indoor combustion of solid fuel is associated with multiple chronic obstructive pulmonary disease (COPD) outcomes. The exposure is associated with both chronic bronchitis and emphysema phenotypes of COPD as well as a distinct form of obstructive airway disease called bronchial anthracofibrosis. COPD from household air pollution differs from COPD from tobacco smoke with respect to its disproportionately greater bronchial involvement, lesser emphysematous change, greater impact on quality of life, and possibly greater oxygen desaturation and pulmonary hypertensive changes. Interventions that decrease exposure to biomass smoke may decrease the risk for incident COPD and attenuate the longitudinal decline in lung function, but more data on exposure–response relationships from well-designed longitudinal studies are needed.

Keywords

emphysema - chronic bronchitis - pulmonary function - bronchial anthracofibrosis - particulate matter