RSS-Feed abonnieren
Bitte kopieren Sie die angezeigte URL und fügen sie dann in Ihren RSS-Reader ein.
https://www.thieme-connect.de/rss/thieme/de/10.1055-s-00000011.xml
Dtsch Med Wochenschr 2009; 134: S419-S421
DOI: 10.1055/s-0029-1243041
DOI: 10.1055/s-0029-1243041
Übersicht | Review article
Angiologie, Intensivmedizin
© Georg Thieme Verlag KG Stuttgart · New York
Welchen Nutzen hat die Selensubstitution in der Post-Reanimationsphase?
Selenium substitution after cardiac arrestWeitere Informationen
Publikationsverlauf
eingereicht: 5.3.2009
akzeptiert: 14.5.2009
Publikationsdatum:
08. Dezember 2009 (online)
![](https://www.thieme-connect.de/media/dmw/2009S11/lookinside/thumbnails/10.1055-s-0029-1243041-1.jpg)
Schlüsselwörter
Natrium-Selenit - oxidativer Stress - Postreanimationssyndrom
Keywords
sodium selenite - oxidative stress - postreanimation syndrome
Literatur
- 1 Adib-Conquy M, Monchi M, Goulenok C. et al . Increased Plasma Levels of Soluble Triggering Receptor Expressed on Myeloid Cells 1 and Procalcitonin after Cardiac Surgery and Cardiac Arrest without Infection. Shock. 2007; 28 406-410
- 2 Adrie C, Adib-Conquy M, Laurent I. et al . Successful cardiopulmonary resuscitation after cardiac arrest as a „sepsis-like” syndrome. Circulation. 2002; 106 562-568
- 3 Adrie C, Laurent I, Monchi M. et al . Postresuscitation disease after cardiac arrest: a sepsis-like syndrome?. Curr Opin Crit Care. 2004; 10 208-212
- 4 Adrie C, Monchi M, Laurent I. et al . Coagulopathy after successful cardiopulmonary resuscitation following cardiac arrest: implication of the protein C anticoagulant pathway. J Am Coll Cardiol. 2005; 46 21-28
- 5 Angstwurm M WA, Engelmann L, Zimmermann T. et al . Selenium in intensive care (SIC) study: Results of a prospective randomized, placebo-controlled, multiple-center study in patients with severe systemic inflammatory response syndrome, sepsis, and septic shock. Crit Care Med. 2007; 35 118-126
- 6 Angstwurm M WA, Schottdorf J, Schopohl J. et al . Selenium replacement in patients with severe systemic inflammatory response syndrome improves clinical outcome. Crit Care Med. 1999; 27 1807-1813
- 7 Arrich J. Clinical application of mild therapeutic hypothermia after cardiac arrest. Crit Care Med. 2007; 35 1041-1047
- 8 Brigelius-Flohé R, Banning A, Schnurr K. Selenium-dependent enzymes in endothelial cell function. Antioxid Redox Signal. 2003; 5 205-215
- 9 Busch H -J, Fink K. et al . Selen-Spiegel in Patienten nach Herz-Kreislaufstillstand nach kardiopulmonaler Wiederbelebung. Intensivmed. 2008; Suppl I , I 57 Poster 06/08
- 10 Carr B G, Goyal M, Band R A. et al . A national analysis of the relationship between hospital factors and post-cardiac arrest mortality. Intensive Care Med. 2008; 35 505-511
- 11 Fink K, Horst C, Busch H J. et al . Massive Freisetzung zirkulierender Endothelzellen und vermehrte endotheliale Apoptose als Zeichen einer schweren Endothelschädigung bei Patienten nach erfolgreicher Reanimation. Clin Res Cardiol. 2008; 97, Suppl 1YIA688 , orale Präsentation
- 12 Fink K, Schwab T, Bode C, Busch H J. Endovascular or surface cooling?: therapeutic hypothermia after cardiac arrest. Anaesthesist. 2008; 57 1155-1160
- 13 Geoghegan M, McAuley D, Eaton S, Powell-Tuck J. Selenium in critical illness. Curr Opin Crit Care. 2006; 12 136-141
- 14 Holzer M, Bernard S A, Hachimi-Idrissi S. et al . Hypothermia for neuroprotection after cardiac arrest: systematic review and individual patient data meta-analysis. Crit Care Med. 2005; 33 414-418
- 15 Laurent I, Adrie C, Vinsonneau C. et al . High-volume hemofiltration after out-of-hospital cardiac arrest: a randomized study. J Am Coll Cardiol. 2005; 46 432-437
- 16 Manzanares Castro W. Selenium in critically ill patients with systemic inflammatory response. Nutr Hosp. 2007; 22 295-306
- 17 McKenzie R C, Arthur J R, Beckett G J. Selenium and the regulation of cell signaling, growth, and survival: molecular and mechanistc aspects. Anitoxid Redox Signal. 2002; 4 339-351
- 18 Neumar R W, Nolan J P, Adrie C. et al . Post-cardiac arrest syndrome: epidemiology, pathophysiology, treatment, and prognostication. A consensus statement from the International Liaison Committee on Resuscitation. Circulation. 2008; 118 2452-2483
- 19 Ozbal S, Erbil G, Kocdor H, Tugyan K, Pekcetin C, Ozogul C. The effects of selenium against cerebral ischemia-reperfusion injury in rats. Neurosci Lett. 2008; 438 265-269
- 20 Parrillo J E. Pathogenetic mechanisms of septic shock. 1993N Engl J Med. 1993; 328 1471-1478
- 21 Pfeifer R, Börner A, Krack A. et al . Outcome after cardiac arrest: predictive values and limitations of the neuroproteins neuron-specific enolase and protein S-100 and the Glasgow Coma Scale. Resuscitation. 2005; 65 49-55
- 22 Sakr Y, Reinhart K, Bloos F. et al . Time course and relationship between plasma selenium concentrations, systemic inflammatory response, sepsis, and multiorgan failure. Br J Anaesth. 2007; 98 775-784
- 23 Zapletal C, Heyne S, Breitkreuz R. et al . The influence of selenium substitution on microcirculation and glutathione metabolism after warm liver ischemia/reperfusion in a rat model. Microvasc Res. 2008; 76 104-109
Dr. med. Hans-Jörg Busch
Kardiologie und Angiologie, Internistische
Intensivtherapie Heilmeyer, Universitätsklinikum
Freiburg
Hugstetterstr. 55
79106 Freiburg
Telefon: 0761/270-3359
eMail: hans-joerg.busch@uniklinik-freiburg.de