Thrombotic microangiopathies

Johanna A. Kremer Hovinga; Bernhard Lämmle

doi:10.1055/s-0037-1619789

Hämostaseologie, Table of Contents

Hamostaseologie 2013; 33(02): 81
DOI: 10.1055/s-0037-1619789

Editorial

Schattauer GmbH

Thrombotic microangiopathies

In honour of Professor Lämmle

Johanna A. Kremer Hovinga MD

,

Bernhard Lämmle Prof.

²Professor of Haematology at the University of Bern and director of the University Clinic of Haematology and Central Haematology Laboratory at the Inselspital in Bern, Switzerland

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Abstract

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References

References
1 Lämmle B, Wuillemin WA, Huber I. et al. Thromboembolism and bleeding tendency in congenital factor XII deficiency – a study on 74 subjects from 14 Swiss families. Thromb Haemost 1991; 65: 117-121.
2 Miyata T, Kawabata S, Iwanaga S. et al. Coagulation factor XII (Hageman factor) Washington D.C. inactive factor XIIa results from Cys-571-Ser substitution. Proc Natl Acad Sci USA 1989; 86: 8319-8322.
3 Miyata T, Kokame K, Matsumoto M, Fujimura Y. ADAMTS13 activity and genetic mutations in Japan. Haemostaseologie 2013; 33: 131-137.
4 Furlan M. Proteolytic cleavage of von Willebrand factor by ADAMTS-13 prevents uninvited clumping of blood platelets. J Thromb Haemost 2004; 02: 1505-1509.
5 Furlan M, Robles R, Lämmle B. Partial purification and characterization of a protease from human plasma cleaving von Willebrand factor to fragments produced by in vivo proteolysis. Blood 1996; 87: 4223-4234.
6 Tsai HM. Physiologic cleavage of von Willebrand factor by a plasma protease is dependent on its conformation and requires calcium ion. Blood 1996; 87: 4235-4244.