Thromb Haemost 1988; 59(02): 340
DOI: 10.1055/s-0038-1642788
Letters to the Editor
Schattauer GmbH Stuttgart

Haemophilia B+ with Inhibitor

M Ludwig
1   Institute of Radiation Biology, Sigmund-Freud-Straβe, 5300 Bonn 1, West Germany
,
R Schwaab
1   Institute of Radiation Biology, Sigmund-Freud-Straβe, 5300 Bonn 1, West Germany
,
K Olek
1   Institute of Radiation Biology, Sigmund-Freud-Straβe, 5300 Bonn 1, West Germany
,
H H Brackmann
2   Institute of Experimental Haematology and Blood Transfusion, Sigmund-Freud-Straβe, 5300 Bonn 1, West Germany
,
H Egli
2   Institute of Experimental Haematology and Blood Transfusion, Sigmund-Freud-Straβe, 5300 Bonn 1, West Germany
› Institutsangaben
Weitere Informationen

Publikationsverlauf

Received 28. September 1987

Accepted 16. November 1987

Publikationsdatum:
21. Mai 2018 (online)

 
  • References

  • 1 Giannelli F, Choo KH, Rees DJ G, Boyd Y, Rizza CR, Brownlee GG. Gene deletions in patients with haemophilia B and anti-factor IXantibodies. Nature 1983; 303: 181-2
  • 2 Matthews RJ, Anson DS, Peake IR, Bloom AL. Heterogeneity of thefactor IX locus in nine hemophilia B inhibitor patients. J Clin Invest 1987; 79: 746-753
  • 3 Brackmann HH, Egli H. Treatment of hemophilia patients withinhibitors. In: Haemophilia. Seligsohn U, Rimon A, Horoszowski H. (eds) Castle House Publications Ltd; London: 1984. pp 113-119
  • 4 Jaye M, De La Salle H, Schamber F, Balland A, Kohli V, Findeli A, Tolstoshev P, Lecocq JP. Isolation of a human anti-haemophilic factorIX cDNA clone using a unique 52-base synthetic oligonucleotide probededuced from the amino acid sequence of bovine factor IX. NucleicAcid Res 1983; 11: 2325-2335