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Thromb Haemost 1994; 72(03): 481-482
DOI: 10.1055/s-0038-1648893
DOI: 10.1055/s-0038-1648893
Original Article
Patients with AT III, Protein C or Protein S Defects Show no Associated Hereditary APC-Resistance
Weitere Informationen
Publikationsverlauf
Received 28. April 1994
Accepted after revision 26. Mai 1994
Publikationsdatum:
25. Juli 2018 (online)
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References
- 1 Svensson PJ, Dahlbäck B. Resistance to activated protein C as a basis for venous thrombosis. N Engl J Med 1994; 330: 517-522
- 2 Koster T, Rosendaal FR, de Ronde H, Briët E, Vandenbroucke JP, Bertina RM. Venous thrombosis due to poor anticoagulant response to activated protein C: Leiden thrombophilia study. Lancet 1993; 342: 1503-1506
- 3 Griffin JH, Evatt B, Wideman C, Fernàndez JA. Anticoagulant protein C pathway defective in majority of thrombophilic patients. Blood 1993; 82: 1989-1993
- 4 Dahlbäck B, Carlsson M, Svensson PJ. Familial thrombophilia due to a previously unrecognised mechanism characterized by poor anticoagulant response to activated protein C: prediction of a cofactor to activated protein C. Proc Natl Acad Sci USA 1993; 90: 1004-1008
- 5 Dahlbäck B, Hildebrand B. Inherited resistance to activated protein C is corrected by anticoagulant cofactor activity found to be a property of factor V. Proc Natl Acad Sci USA 1994; 91: 1396-1400
- 6 Halbmayer W-M, Haushofer A, Schon R, Fischer M. The prevalence of poor anticoagulant response to activated protein C (APC resistance) among patients suffering from stroke or venous thrombosis and among healthy subjects. Blood Coagul and Fibrinol 1994; 5: 51-57