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Thromb Haemost 1985; 54(04): 901
DOI: 10.1055/s-0038-1660163
DOI: 10.1055/s-0038-1660163
Letter to the Editor
Dihomo-γ-Linolenic Acid in Patients with Atherosclerosis: Effects on Platelet Aggregation, Plasma Lipids and Low-Density Lipoprotein-Induced Inhibition of Prostacyclin Generation
Weitere Informationen
Publikationsverlauf
Received 03. Dezember 1984
Accepted 16. September 1985
Publikationsdatum:
19. Juli 2018 (online)
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References
- 1 Szczeklik A, Gryglewski RJ, Sladek K, Kostka-Trabka E, Zmuda A. Dihomo-y-linolenic acid in patients with atherosclerosis: effects on platelet aggregation, plasma lipids and low-density lipoprotein-induced inhibition of prostacyclin generation. Thromb Haemostas 1984; 51: 186-188
- 2 Mikhailidis DP, Kirtland SJ, Barradas MA, Dandona P. Dihomogammalinolenic acid inhibits platelet aggregation and stimulates platelet prostaglandin production in healthy subjects: absence ofeffects in insulin-dependent diabetics. Diabetologia 1984; 27: 311A
- 3 Hutton RA, Mikhailidis DP, Bernstein RM, Jeremy JY, Hughes GR V, Dandona P. Assessment of platelet functionin patients with Raynaud’s syndrome. J Clin Pathol 1983; 37: 182-187
- 4 Mikhailidis DP, Mikhailidis AM, Barradas MA, Dandona P. Effect of nonesterified fatty acids on the stability of prostacyclin activity. Metabolism 1983; 32: 717-721
- 5 Brox JH, Nordoy A. The effect of polyunsaturated fatty acids on endothelial cells andtheir production of prostacyclin, thromboxane and platelet inhibitory activity. Thromb Haemostas 1983; 50: 762-767
- 6 Dandona P, Lomeo F, Khokher MA, Houlder S. Dihomogammalinolenic acid (DHLA) and linolenic acid potentiate thestimulatory effect of insulin and adipocyte lipogenesis. Diabetes 1983; 32 (Suppl. 01) 60A