Thromb Haemost 1984; 51(01): 103-104
DOI: 10.1055/s-0038-1661031
Original Article
Schattauer GmbH Stuttgart

Response of Femoral Venous Oxygen Tension to Graduated Pressure Stockings - Possible Relationship to Deep Vein Thrombosis

G C Okoye
The Bioengineering Unit, University of Strathclyde, Glasgow and the University Departments of Medicine and Medical Cardiology, Royal Infirmary, Glasgow, Great Britain
,
J H Evans
The Bioengineering Unit, University of Strathclyde, Glasgow and the University Departments of Medicine and Medical Cardiology, Royal Infirmary, Glasgow, Great Britain
,
J Beattie
The Bioengineering Unit, University of Strathclyde, Glasgow and the University Departments of Medicine and Medical Cardiology, Royal Infirmary, Glasgow, Great Britain
,
G D O Lowe
The Bioengineering Unit, University of Strathclyde, Glasgow and the University Departments of Medicine and Medical Cardiology, Royal Infirmary, Glasgow, Great Britain
,
A R Lorimer
The Bioengineering Unit, University of Strathclyde, Glasgow and the University Departments of Medicine and Medical Cardiology, Royal Infirmary, Glasgow, Great Britain
,
C D Forbes
The Bioengineering Unit, University of Strathclyde, Glasgow and the University Departments of Medicine and Medical Cardiology, Royal Infirmary, Glasgow, Great Britain
› Author Affiliations
Further Information

Publication History

Received 04 November 1983

Accepted 01 December 1983

Publication Date:
19 July 2018 (online)

Summary

Venous oxygen tension (pO2) was measured in discrete samples of blood obtained through the femoral vein of cardiac catheterisation patients before, during and after application of sustained external graduated pressure in the form of compression stockings (T. E. D. Kendall). There was a significant reduction (p <0.05) of pO2 from the baseline value both 30 sec and one minute after the application of the pressure stockings. Thereafter, the pO2 rose to baseline values. Two minutes after the stockings were removed there was again a significant reduction (p <0.05) of pO2 from the baseline value. We suggest that the decrease in ivenous pO2 on application of external pressure may be a reflection of washing out of stagnant hypoxic blood from the venous valve pockets, which may be related to the formation of deep vein thrombosis (DVT).

 
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