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Thromb Haemost 1997; 78(06): 1528-1529
DOI: 10.1055/s-0038-1665447
DOI: 10.1055/s-0038-1665447
Letter to the Editor
The Endothelial von Willebrand Factor in Venous Occlusion Blood
Further Information
Publication History
Received 07 1997
Accepted after resubmission 08 August 1997
Publication Date:
12 July 2018 (online)
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References
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- 2 Wieczoreck I, Ludlam CA, MacGregor I. Venous occlusion does not release von Willebrand factor factor VIII or PAI-1 from endothelial cells – the importance of consensus on the use of correction factors for haemoconcentration. Thromb Haemost 1993; 69: 91-93
- 3 Scharrer I, Vigh Z. Release of von Willebrand factor after venous occlusion – the importance of standardization of venous occlusion tests. Thromb Haemost 1993; 69: 880-881
- 4 Wieczorek I, Ludlam CA. Rebbuttal to the letter to the editor by Dr Scharrer et al. Thromb Haemost 1993; 69: 881
- 5 Alessi MC, Boffa MC, Menart C, Aillaud MF, Parrot G, Juhan-Vague I. Venous occlusion does not induce the release of thrombomodulin from endothelial cells in patients with thromboembolic disease. Thromb Haemost 1992; 68: 483-484
- 6 Keber D. On the use of different correction factors for hemoconcentration. Thromb Haemost 1983; 049-245
- 7 Moake JR, Turner NA, Stathopoulos NA, Nolaasco LH, Heliums JD. Involvement of large plasma von Willebrand factor (vWF) multimers and unusually large vWF forms derived from endothelial cells in shear stress- induced platelet aggregation. J Clin Invest 1986; 78: 1456-1461
- 8 O’Brien JR, Salmon GP. Shear stress activation of platelet glycoprotein IIb/IIIa plus von Willibrand factor causes aggregation. Blood 1987; 70: 1354-1361
- 9 O’Brien JR, Etherington MD, Salmon GP. The diagnosis of glycoprotein (GP) IIb/IIIa deficiency differentiated from vWF and GPIb deficiency using a high shear filter device. Abstr 2146 Thromb Haemost 1993; 69: 1142