Thromb Haemost 2015; 113(05): 915
DOI: 10.1160/TH15-03-0218
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Localising oxidant “stress” in the antiphospholipid syndrome

Fred S. Lamb
1   Department of Pediatrics, Vanderbilt University Medical Center, Nashville, Tennessee, USA
› Institutsangaben
Weitere Informationen

Publikationsverlauf

Received: 11. März 2015

Accepted 11. März 2015

Publikationsdatum:
24. November 2017 (online)

 
  • References

  • 1 Giannakopoulos B, Krilis SA. The pathogenesis of the antiphospholipid syndrome. N Engl J Med 2013; 368: 1033-1044.
  • 2 Spencer NY, Engelhardt JF. The basic biology of redoxosomes in cytokine-mediated signal transduction and implications for disease-specific therapies. Biochemistry 2014; 53: 1551-1564.
  • 3 Lassegue B, Griendling KK. NADPH Oxidases: Functions and Pathologies in the Vasculature. Arterioscler Thromb Vasc Biol 2009; 30: 653-661.
  • 4 Mueller-Calleja N, Köhler A, Siebald B. et al. Cofactor-independent antiphospholipid antibodies activate the NLRP3-inflammasome via endosomal NADPH-oxidase: implications for the antiphospholipid syndrome. Thromb Haemost 2015; 113: 1071-1083.