Intramuscular Abscesses
Infection and subsequent inflammation of muscle may be due to varied etiology, such
as hematogenous septic seeding (bacterial, fungal), direct extension from a local
source of infection (i.e., osteomyelitis, cellulitis), or via a parallel autoimmune
response to remote infection, such as in Lyme disease and influenza. The most common
microbe implicated is Staphylococcus aureus, and in a Koch’s endemic country like India, Mycobacterium tuberculosis.
On ultrasonography (USG), abscesses typically appear as variably defined heteroechoic
collections, with internal debris and occasional septation. Enlargement of the affected
muscle is seen. Perilesional vascularity is marked on color Doppler, and edema with
inflammatory changes is noted to surround the lesion. On computed tomography (CT),
enlargement of the muscle with surrounding fat-stranding is noted. The abscess per
se appears as a focal hypodensity with peripheral rim enhancement with contrast, which
helps differentiate it from myonecrosis, which does not show enhancement. On magnetic
resonance (MR) [Figure 1], muscular enlargement with edema is present as T2 hyperintensity. The abscess appears
T1-hypointense and T2-hyperintense, with peripheral contrast enhancement and occasional
internal septation and fluid–fluid level [Figure 2]A and [Figure 2]B. However, signal intensity may vary depending on the proportion of blood/proteinaceous
material present within the cavity. This may cause confusion in the differentiation
of abscesses and hematomata.
Figure 1: Femoral osteomyelitis in a 40-year-old male with left thigh pain. Axial STIR MR image
of bilateral thighs shows left proximal medullary femoral hyperintensity with spread
into the adductor and hamstring muscles. Incidental STIR hyperintensity of the right
gluteus maximus noted after intramuscular injection administration
Figure 2 (A and B): Abscess within the quadriceps in a 20-year-old immunocompromised female. (A) Sagittal
T1W and (B) sagittal T2W images of the thigh showing fluid-fluid level in a suppurative
collection within the vastus intermedius
Though abscesses intuitively appear to present as acute collections, they may mimic
a malignant mass lesion in the subacute or chronic clinical scenario. In such cases,
cold abscesses caused by M. tuberculosis are to be considered. Occurring as a consequence to unmitigated infection of the
vertebrae (i.e., Pott disease) [Figure 3] and [Figure 4], these muscular collections often coexist with destructive vertebral lesions and
other stigmata of Koch’s. On imaging, they are seen to appear similar to their acute
counterparts.
Figure 3: Cold abscess in a 42-year-old male with active pulmonary Koch's. Axial contrast-enhanced
(CE) CT image shows a well-defined, peripherally enhancing hypodensity in the left
psoas, crossing the midline. Aspirates revealed acid fast bacilli (AFB)
Figure 4: Cold abscess in a 67-year-old lady with lower back ache. Coronal MRI shows a T2/STIR
hyperintense collection noted within the right psoas muscle, noted to extend from
the L1-L2 intervertebral disc. Histopathology confirmed the presence of AFB. Incidentally
noted is a right renal cyst
Intramuscular Benign Tumors
Lipoma
Lipomata are the most common benign mesenchymal soft-tissue tumors. Intramuscular
lipomata are uncommon and constitute just over 1.8% of all primary tumors of adipose
tissue and <1% of all lipomas.[1] On ultrasound (US), they are usually well-defined, hyperechoic masses with scanty
internal vascularity.[2]
On CT [Figure 5], lipomata are usually well-defined and homogeneous, with a low tissue attenuation
(approx. –65 to –120HU), without contrast enhancement. On MR imaging (MRI) [Figure 6]A, [Figure 6]B, [Figure 6]C, [Figure 6]D, the signal intensity of lesion parallels that of subcutaneous fat on all sequences,
being hyperintense to skeletal muscle on both T1- (T1W) and T2-weighted (T2W) images.
Figure 5: Lipoma incidentally discovered in a 22-year-old male. Axial CECT image shows a left
gluteal intramuscular lipoma showing fat attenuation with no enhancement
Figure 6 (A-D): Lipoma in a 30-year-old male with chronic, painless calf swelling. Coronal (A) T1W
hyperintense, (B) T2W hyperintense, (C) sagittal STIR hypointense well-defined lesion
within the medial head of gastrocnemius, exhibiting (D) fat suppression with minimal
septal enhancement on sagittal CE T1W image
Hemangioma
Hemangiomata frequently involve soft tissue. Soft-tissue hemangiomata are benign endothelial
neoplasms that histologically resemble normal blood vessels. Intramuscular hemangiomata
account for approximately 0.8% of all benign soft-tissue tumors.[3] US can identify abnormal Doppler flow patterns or features consistent with phleboliths.
Angiography may demonstrate the fine vascular details of a hemangioma, useful when
embolization or surgical resection of complex lesions is considered. Presence of fat
within a hemangioma is characteristic, and often serves to differentiate it from an
intramuscular arteriovenous malformation (the latter of which tends to exhibit phleboliths).
On MRI [Figure 7]A, [Figure 7]B, [Figure 7]C, T1W images show an amorphous low-to-intermediate signal intensity mass with ill-defined
margins due to peripheral flow voids and variable intralesional fat suppression [Figure 8]A, [Figure 8]B and [Figure 9]A, [Figure 9]B. T2W images show low-signal intensity in areas of vascular components. Plain CT
[Figure 7]D generally shows a poorly defined lesion with attenuation similar to that of skeletal
muscle.
Figure 7 (A-D): Hemangioma in a 30-year-old male with painless swelling in the right thigh. (A) Coronal
T1W shows a heterogeneously hyperintense lesion within right vastus lateralis; (B)
Axial T2W and (C) axial STIR images show a heterogeneously hyperintense lesion; (D)
Axial unenhanced CT image shows a hypoattenuating lesion
Figure 8 (A and B): Hemangioma in a 37-year-old female with right quadriceps weakness. (A) Axial STIR
image shows a heterogeneously hyperintense intramuscular focus in the right quadriceps,
with intralesional areas of fat suppression; (B) Sagittal T2W image shows an ill-defined
intramuscular hyperintensity
Figure 9 (A and B): Hemangioma in a 20-year-old male with painless swelling of the posterior compartment
of arm. (A) Coronal STIR image shows a hyperintense lesion within the lateral head
of the triceps; (B) Coronal T1W image shows a hypointense mass (arrow)
Neurofibroma
Neurofibromata [Figure 10]A and [Figure 10]B are benign peripheral nerve sheath tumors, usually solitary and sporadic. A majority
of cases are localized (90%), and not associated with neurofibromatosis Type 2.[4] They may also be diffuse or plexiform, the latter being pathognomonic of neurofibromatosis
Type 1. On CT, localized neurofibromata are well-defined, hypodense, and show little-to-no
contrast enhancement. On MR, they appear T1-hypointense, T2-hyperintense, and show
heterogeneous contrast enhancement.
Figure 10 (A and B): Neurofibroma in a 60-year-old female with known orbital neurofibromatosis. (A) Axial
T1W shows an isointense lesion (arrow) within the right temporalis muscle; (B) Coronal CE T1W image shows the lesion's heterogeneous
enhancement
Desmoid Fibromatosis
Desmoid fibromatoses [Figure 11]A and [Figure 11]B are a rare (incidence of 0.03% of all neoplasms [5]) broad group of fibroproliferative lesions that show myofibroblastic cells, dense
collagen deposition, and variable myxoid stroma. Aggressive desmoid fibromas show
local infiltrative growth and recurrence, but are unable to metastasize. They may
occur in the fascial or musculo-aponeurotic planes. On USG, they have smooth, sharp
margins with variable echogenicity; unenhanced CT may show a well-defined hyperattenuating
intramuscular lesion with variable contrast enhancement. MR reveals hypointensity
on T1 and T2 weighting, with moderate to marked contrast enhancement.
Figure 11 (A and B): Desmoid fibromatosis in a 56-year-old male with progressive weakness of the extensor
compartment of right thigh. (A) Unenhanced axial CT image of the lower limbs shows
a well-defined mass within the right vastus intermedius with extension to the vastus
lateralis; (B) CE axial CT image shows the lesion's heterogeneous enhancement
Myxoma
Intramuscular myxomas [Figure 12]A, [Figure 12]B, [Figure 12]C are a rare benign type of soft-tissue myxoma, which present as deep-seated masses
composed of fibroblasts and abundant myxoid stroma. With an incidence of approximately
0.1 per 100,000, the average age of detection is 40–60 years predominantly in female
patients.[6] USG reveals a hypoechoic mass with a heterogeneous echotexture, often with posterior
acoustic enhancement. On MR, it appears T1-hypointense, T2-hyperintense, and shows
variable peripheral and internal contrast enhancement.[7]
Figure 12 (A-C): Myxoma in a 48-year-old male with progressive left groin swelling. (A) Coronal T1W
image shows a hypointense, well-defined lesion within the left adductor magnus; (B)
Coronal T2W image shows a hyperintense mass; (C) CE coronal T1W image reveals the
lesion's heterogeneous enhancement
Hamartoma
Mesenchymal hamartomata [Figure 13]A, [Figure 13]B, [Figure 13]C, [Figure 13]D are usually benign collection of heterotopically located mature soft tissue. They
may contain muscular, lipomatous, fibrous, and neural components.
Figure 13 (A-D): Hamartoma in a 27-year-old female with pain in the medial compartment of left thigh.
(A) Coronal T1W image shows a well-defined iso-hypointense lesion within the left
vastus medialis; (B) Axial STIR image shows hyperintensity; (C) Axial CT reveals isointensity
with speck of central calcification (arrow); (D) Ultrasound shows a vascular isoechoic lesion
US reveals a homogeneously isoechoic, vascular lesion. On MRI, the lesion appears
T1 iso-to-hypointense, and T2 and short inversion time inversion recovery (STIR) hyperintense.
Heterogeneous enhancement is seen postcontrast. CT shows a well-defined hypodensity.
Typically, biopsy reveals fibromuscular and adipose tissue showing many proliferating,
dilated, congested, thick, and thin-walled blood vessels, along with intramuscular
adipose tissue, smooth muscle, and neural tissue, suggestive of a mesenchymal hamartoma.
Intramuscular Malignant Tumors
Soft-tissue sarcomas are a heterogeneous group of tumors that arise from tissue of
mesenchymal origin and are characterized by infiltrative local growth.
Primary intramuscular malignant tumors
Rhabdomyosarcoma
Rhabdomyosarcomas are the most common soft-tissue tumor in children, and commonly
occur in adults under the age of 45. On US [Figure 14]D, muscular sarcomas (rhabdomyosarcomas) appear as an irregular, usually well-defined
mass with low to intermediate echogenicity. Marked vascularity may be noted on color
Doppler imaging.
On CT, these masses show soft-tissue attenuation with variable contrast enhancement.
On MRI [Figure 14]A, [Figure 14]B, [Figure 14]C, they appear T1-hypointense and T2-hyperintense, with variable contrast enhancement.
Foci of necrosis within the tumor herald a poor prognosis.
Figure 14 (A-D): Rhabdomyosarcoma in a 21-year-old male with rapidly progressive, painful swelling
of the left thigh. (A) Coronal T1W image shows isointense lesion within left vastus
lateralis; (B) Sagittal T2W image shows a hyperintense lesion, with necrosis; (C)
Axial STIR image shows hyperintensity; (D) Hypoechogenicity with vascularity on US
Diffuse large B-cell lymphoma
Diffuse large B-cell lymphoma (DLBCL) is the most prevalent form of non-Hodgkin’s
lymphoma (NHL), and has a high incidence among the elderly, with a predilection for
involving the muscles surrounding the pelvic girdle. Primary involvement of skeletal
muscle by DLBCL is exceptionally rare, and accounts for 0.5% of all extranodal lymphomas.[8] US of the same may reveal diffusely bulky muscle, and may sometimes show a heterogeneous,
hypoechoic variably defined mass in the substance of the muscle. On CT [Figure 15]A and [Figure 15]B, the affected muscle(s) appears bulky and shows diffuse contrast enhancement. MRI
[Figure 16] is considered superior in imaging of primary muscular lymphomas. Isointensity to
muscle on T1W images and hyperintensity on T2W images are seen.
Figure 15 (A and B): Primary muscular B-cell lymphoma in a 64-year-old female with progressive swelling
and weakness of the left thigh. (A) Unenhanced axial CT image reveals a large iso-attenuating
lesion in the intramuscular plane of the posterior thigh; (B) CECT images show heterogeneous
enhancement of the lesion
Figure 16: Primary muscular non-Hodgkin's lymphoma in a 67-year-old female with nonspecific
abdominal pain. Coronal STIR image shows diffusely bulky and hyperintense right iliopsoas
and gluteus maximus
Atypical lipomatous tumors
Atypical lipomatous tumors (ALT) is the new term that has replaced the older “liposarcoma”
(according to the World Health Organization classification of soft-tissue tumors in
2013[9]) [Figure 17]A and [Figure 17]B. They are the second most common soft-tissue sarcoma, accounting for 10–35% of
all cases. Lesions are commonly intramuscular, and involve the lower extremity (i.e.,
thigh) more frequently than the upper. On US, a heterogeneous, multilobulated, typically
well-defined mass is noted. CT and MR show a well-defined, lobulated, predominantly
fat-intensity lesion with foci of nonadiposity within. Nonlipomatous components are
most often seen as prominent thick septa (>2 mm) that may show nodularity, and this
serves to differentiate it from a lipoma. Moderate to marked enhancement of the septa
is noted with contrast.
Figure 17 (A and B): ALT in a 52-year-old female with progressive left thigh swelling and neuropathic
pain. (A) Coronal T1W image shows a lobulated, well-defined fat-intensity lesion within
the left vastus intermedius; (B) Coronal STIR image shows fat signal attenuation within
the lesion with muscular edema
Secondary intramuscular malignant tumors
Metastases to skeletal muscle are rare, and are seen in 16–17.5% of all autopsies
of cancer patients with widespread metastatic disease, predominantly in the musculature
of the trunk. The common primaries involved are genital tumors (24.6%), gastrointestinal
tumors (21.3%), urological tumors (16.4%), and malignant melanoma (13.1%), bronchial
carcinoma (8.2%), thyroid gland carcinoma (4.9%), and breast carcinoma (3.3%).[10],[11]
On US, muscular metastases are noted to be variably defined, hypoechoic masses within
the substance of the muscle. The CT appearance [Figure 18]A and [Figure 18]B is usually that of hypoattenuating lesions with variable definition and contrast
enhancement.
Figure 18 (A and B): Muscular metastasis in a 70-year-old male with unknown primary. (A) CE axial CT image
and (B) sagittal reconstruction image show multiple ill-defined enhancing lesions
within a bulky insertion of the left iliopsoas
On MRI [Figure 19]A and [Figure 19]B, skeletal metastases appear isointense to muscle on T1 images, show heterogeneous
signal with perilesional edema on T2 images, and show peripheral enhancement on contrast
images [Figure 20]A, [Figure 20]B, [Figure 20]C. A rare tumor to metastasize muscle is synovial sarcoma. Despite its name, synovial
sarcoma is not of synovial origin, and is usually extraarticular. Common occurrence
is within the soft tissue around the joints of the extremities (lower limb commonly)
[Figure 21]A, [Figure 21]B, [Figure 21]C, [Figure 21]D. The ultimate diagnosis of a suspected muscular metastasis, however, is via histopathology.
Figure 19 (A and B): Muscular metastasis in a 44-year old female with breast carcinoma. (A) Coronal STIR
image shows diffusely bulky. hyperintense muscles of the left infraspinatus and supraspinatus.
representing muscular metastasis; (B) Coronal STIR image of abdomen reveals hyperintensities
within the liver representing metastases
Figure 20 (A-C): Muscular metastasis in a 21-year-old male with Ewing's sarcoma. (A) Coronal T1W image
shows a hypointense lesion in left latissimus dorsi (arrow); (B) Coronal T2W image
shows mild hyperintensity; (C) CE T1W image shows peripheral enhancement. Also noted
is involvement of left iliac bone
Myositis ossificans
Myositis ossificans (MO) is the occurrence of heterotopic ossification that usually
follows trauma to the associated joint. It may also occur without history of trauma,
in paraplegics. MO circumscripta is heterotopic calcification that follows trauma,
whereas MO progressiva (now known as fibrodysplasia ossificans progressiva) is a rare
inherited disorder inherited predominantly via sporadic mutation.
Myositis ossificans circumscripta
MO circumscripta on radiographs appear as peripherally ossified, centrally lucent
masses usually found around joints. Calcification usually starts by approximately
2 weeks, and assumes its classical appearance by 2 months. CT findings [Figure 22]A are similar, demonstrating calcification that proceeds from the periphery to the
center.
Figure 21 (A-D): Synovial sarcoma of foot with metastasis in a 57-year-old male. (A) Sagittal STIR:
hyperintense lesion involving forefoot; (B) Coronal T1W image thigh: iso-hyperintense
metastasis (arrow) involving adductor magnus; (C) Coronal T2W image reveals hyperintensity; (D) Sagittal
CE T1W FatSat image: enhancement with central necrosis
Figure 22 (A-D): Myositis ossificans circumscripta in a 33-year-old female with history of elbow trauma.
(A) Axial CT shows peripheral calcification (arrow); (B) Coronal T1W, and (C) Coronal
gradient echo images show periarticular mineralization; (D) Axial STIR shows heterogeneous
hyperintensity
On MRI [Figure 22]B, [Figure 22]C, [Figure 22]D, features vary with the age of the lesion. Early MO is T1 isointense to muscle,
T2 hyperintense peripherally, and heterogeneously hyperintense centrally. Late MO
resembles bone and shows low peripheral and high central intensity on both sequences.
While early MO may heterogeneously enhance, late MO usually does not.
Myositis ossificans progressiva
MO progressiva [Figure 23] is characterized by insidious fibrosis and calcification of muscles, ligaments,
and tendons that ultimately proves fatal due to involvement of the respiratory musculature.
Figure 23: Myositis ossificans progressiva (fibrodysplasia ossificans progressiva) in a 26-year-old
male with progressive breathing difficulty. Unenhanced axial CT of the chest reveals
extensive muscular ossification involving the left anterior chest and upper limbs.
Band-like ossification is noted involving the interscapular region
Intramuscular hematomata
Intramuscular hematomata may be spontaneous, or secondary to hemorrhagic diathesis,
anticoagulant therapy, trauma, tumor, recent surgery, or biopsy. In muscular trauma,
blunt injury is commonly implicated. US imaging of acute mild muscular trauma may
reveal focal isoechoic muscle swelling against the background of the undamaged muscle
tissue. The appearance of more severe contusions with hematoma formation varies with
the elapsed time: within 24 hours, hematomata may appear both hypo- and hyperechoic.
In the following days, hematomas may appear hypo- or anechoic, until coagulation renders
them inhomogeneous. Ultimately, they resolve, sometimes with residual scarring. In
case of incisive injuries, US may reveal disrupted muscle fibers with local hematoma
[Figure 24]A.
Figure 24 (A-C): Hematoma in a young male with history of trauma to the abdominal wall. (A) Heteroechoic
psoas hematoma on ultrasound; (B) Unenhanced axial CT image shows heterodense collections
within the right psoas muscle, and anterolateral abdominal musculature; (C) Axial
CECT of the pelvis shows lack of enhancement
CT images [Figure 24]B and [Figure 24]C show increased bulk of the affected muscle, with intramuscular hyperdensity (>40
HU), representing acute bleed. As it develops, a focal hypodensity takes its place.
MR appearance [Figure 25]A and [Figure 25]B varies with the age of the hematoma: acute hematomata show iso-hypointensity to
muscle on T1W; and slightly hypo- or hyperintensity on T2W. Subacute hematomata show
three levels of intensity on T1 images: a low-intensity capsular sign, high-intensity
in the peripheral zone, and central isosignal.
Figure 25 (A and B): Subacute hematoma in an 18-year-old male with hemophilia. (A) Axial T1W image and
(B) Axial T2W image of the pelvis shows a heterointense collection (arrow) near the insertion of the right iliacus
As the hematoma develops, the central and peripheral signal intensities tend to decrease,
on both T1W and T2W sequences. Chronic hematomas may present with hypointense collections
on T1W images and hyperintense on T2W images.[12] In all cases of suspected hematoma, clinical/sonological follow-up must be done
till resolution, in order to conclusively rule out a hemorrhagic sarcoma.
Intramuscular vascular malformations
Vascular malformations need to be differentiated from true vascular neoplasms such
as hemangiomata, as this holds therapeutic and prognostic significance. Vascular malformations
are classified on the basis of MRI as being either high-flow malformations [arteriovenous
(AV) malformations, AV fistulae], or slow-flow malformations (venous, lymphatic, capillary,
or combined).
On US, venous malformations appear as multiple compressible dilated tortuous veins
clumped together. Color Doppler flow [Figure 26]A and venous waveform should be sought within this lesion, which, if present, is
diagnostic. Changes in flow are noted with maneuvers such as gravity dependency and
Valsalva. However, due to sluggish flow, flow may be absent altogether. Phleboliths
may be seen [Figure 27], which is often the distinguishing characteristic between an arteriovenous malformation
and a hemangioma, the latter of which tends to contain intralesional fat. On MRI,
the slow-flow channels are seen as T2-hyperintense lobules or serpiginous channels.
Contrast MR shows delayed, heterogeneous enhancement [Figure 26]B and [Figure 26]C, [Figure 28]A, [Figure 28]B, [Figure 28]C.
Figure 26 (A-C): Slow-flow vascular malformation in lateral head of gastrocnemius in a 34-year-old
male with chronic, progressive, painless calf swelling. (A) Marked lesion vascularity
on Doppler; (B) Coronal T1W image reveals an isointense lesion; (C) Coronal STIR image
shows hyperintensity
Figure 27: Slow-flow venous malformation in the belly of right gastrocnemius in a 38-year-old
male with occasional dragging calf pain. Axial unenhanced CT of the leg shows an iso-to-hypodense
lesion with phleboliths, characteristic of arteriovenous malformation. Hemangiomata,
in contrast, tend to contain intralesional fat
Figure 28 (A-C): Slow-flow venolymphatic malformation of the left vastus lateralis and glutei in a
30-year-old female with soft, compressible lateral thigh swelling. (A) Coronal T1W
image of the left lower limb shows a large isointense lesion; (B) Coronal STIR and
(C) axial T2W images show hyperintensity
Myocysticercosis
Cysticercosis is a parasitic infection caused by ingestion of the eggs of Taenia solium or Taenia saginata, transmitted through the feco-oral route. Humans are a definitive host for the adult
parasite. Cysticerci are the larval stage of the tapeworm, and may be found disseminated
in neural and soft tissue, where it incites an inflammatory reaction and may calcify
over extended periods of time. The clinical differential diagnosis for soft-tissue
cysticercoses may be lipoma, epidermoid cyst, abscess/pyomyositis, tuberculous lymphadenitis,
myxoma, neurofibroma, or fat necrosis.
Diagnosis may be made on the basis of radiographs, which may show multiple calcified
specks (so-called “rice-grain” calcification) in the substance of muscle. US may reveal
a well-defined lesion with surrounding perilesional edema, often revealing the parasite’s
scolex within [Figure 29].
Figure 29: Myocysticercosis in a 30-year-old male with chronic calf swelling and tenderness.
Ultrasound image shows a well-defined ovoid intramuscular lesion with perilesional
edema and a scolex within
On MR [Figure 30]A and [Figure 30]B, it appears hypointense on T1W and hyperintense on T2W images. Peripheral rim enhancement
of the cyst wall may be seen. Intramuscular cysts are oriented in the direction of
the muscle fibers. The scolex may be seen as a tiny hypointense speck within the hyperintense
cyst.[13] Presence of such a lesion in a patient with known neurocysticercosis should always
raise suspicion for myocysticerosis.
Figure 30 (A and B): Myocysticercosis in a 20-year-old female with neurocysticercosis and recurrent episodes
of seizure. (A) Axial T2 FLAIR image of the brain reveals multiple parenchymal hypointensities
with eccentric hyperintense scolices; (B) Coronal T2 image shows similar lesions in
the extraocular muscles, muscles of mastication and tongue
Intramuscular endometriosis
Endometriosis is the presence of endometrial tissue outside the uterine cavity. Abdominal
wall endometriosis may occur following the implantation of endometrial cells into
the soft tissues of the abdominal wall after open uterine surgeries like cesarean
sections. The incidence rate is reported at 0.4–0.1%.[14]
US [Figure 31]A reveals a heterogeneous hypoechoic, irregularly marginated lesion within the affected
muscle (commonly the abdominal recti). Color Doppler may reveal flow. On CT [Figure 31]B, irregularity and bulkiness of the affected muscle may be seen, with occasional
hyperdense/hypodense foci within, representing the endometrial islands. MR reveals
a poorly contrast-enhancing heterogeneous hypo-hyperintense area within the affected
muscle, with muscle bulkiness.
Figure 31 (A and B): Intramuscular endometriosis in a 30-year-old female with past history of cesarean
section. (A) Ultrasound reveals an isoechoic intramuscular island; (B) Axial CECT
of the pelvis shows the lesion within an irregularly enlarged left rectus abdominis,
with heterogeneous enhancement
