Erfahrungsbericht zum Projekt Untersuchung der Regulierung axonaler Exzitabilität in peripheren sensorischen und motorischen Axonen mittels der „Threshold tracking“ – Methode ^[*]

• Literatur

• 1 Krishnan AV, Lin CS, Park SB et al. Axonal ion channels from bench to bedside: a translational neuroscience perspective. Prog Neurobiol 2009; 89: 288-313
• 2 Bostock H, Cikurel K, Burke D. Threshold tracking techniques in the study of human peripheral nerve. Muscle Nerve 1998; 21: 137-158
• 3 Krishnan AV, Lin CS, Kiernan MC. Activity-dependent excitability changes suggest Na+/K+ pump dysfunction in diabetic neuropathy. Brain 2008; 131: 1209-1216
• 4 Lin CS, Krishnan AV, Lee MJ et al. Nerve function and dysfunction in acute intermittent porphyria 2008; 131: 2510-2519
• 5 Krishnan AV, Goldstein D, Friedlander M et al. Oxaliplatin-induced neurotoxicity and the development of neuropathy. Muscle Nerve 2005; 32: 51-60
• 6 Kiernan MC, Isbister GK, Lin CS et al. Acute tetrodotoxin-induced neurotoxicity after ingestion of puffer fish. Ann Neurol 2005; 57: 339-348
• 7 Lin CS, Macefield VG, Elam M et al. Axonal changes in spinal cord injured patients distal to the site of injury. Brain 2007; 130: 985-994
• 8 Tomlinson SE, Tan SV, Kullmann DM et al. Nerve excitability studies characterize Kv1.1 fast potassium channel dysfunction in patients with episodic ataxia type 1. Brain 2010; 133: 3530-3540
• 9 Krishnan AV, Bostock H, Ip J et al. Axonal function in a family with episodic ataxia type 2 due to a novel mutation. J Neurol 2008; 255: 750-755
• 10 Kiernan MC, Krishnan AV, Lin CS et al. Mutation in the Na+ channel subunit SCN1B produces paradoxical changes in peripheral nerve excitability. Brain 2005; 128: 1841-1846