Aktuelle Neurologie, Inhaltsverzeichnis Aktuelle Neurologie 2012; 39(09): 486-487DOI: 10.1055/s-0032-1327290 Kompetenznetz Multiple Sklerose © Georg Thieme Verlag KG Stuttgart · New York Aktuelles aus der Forschung T. Menge 1 Neurologische Klinik, Heinrich-Heine-Universität, Düsseldorf , G. Meyer zu Hörste 1 Neurologische Klinik, Heinrich-Heine-Universität, Düsseldorf 2 Center for Neurologic Diseases, Brigham and Women’s Hospital, Harvard Medical School, Boston, MA, USA › Institutsangaben Artikel empfehlen Abstract Artikel einzeln kaufen Volltext Referenzen Literatur 1 Quintana FJ, Farez MF, Izquierdo G et al. Antigen microarrays identify CNS-produced autoantibodies in RRMS. Neurology 2012; 78: 532-9 2 Elliott C, Lindner M, Arthur A et al. Functional identification of pathogenic autoantibody responses in patients with multiple sclerosis. Brain 2012; 135: 1819-1833 3 Comabella M, Fernandez M, Martin R et al. Cerebrospinal fluid chitinase 3-like 1 levels are associated with conversion to multiple sclerosis. Brain 2010; 133: 1082-1093 4 Canto E, Reverter F, Morcillo-Suarez C et al. Chitinase 3-like 1 plasma levels are increased in patients with progressive forms of multiple sclerosis. Mult Scler 2012; 18: 983-990 5 Bai L, Lennon DP, Eaton V et al. Human bone marrow-derived mesenchymal stem cells induce Th2-polarized immune response and promote endogenous repair in animal models of multiple sclerosis. Glia 2009; 57: 1192-1203 6 Bai L, Lennon DP, Caplan AI et al. Hepatocyte growth factor mediates mesenchymal stem cell-induced recovery in multiple sclerosis models. Nat Neurosci 2012; 15: 862-870 7 Hirota K, Duarte JH, Veldhoen M et al. Fate mapping of IL-17-producing T cells in inflammatory responses. Nat Immunol 2011; 12: 255-263 8 Lee Y, Awasthi A, Yosef N et al. Induction and molecular signature of pathogenic T(H)17 cells. Nat Immunol 2012; 13: 991-999
