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Thromb Haemost 2000; 84(04): 724-725
DOI: 10.1055/s-0037-1614095
DOI: 10.1055/s-0037-1614095
Letters to the Editor
The C536T Transition in the Tissue Factor Pathway Inhibitor (TFPI) Gene Does not Contribute to Risk of Venous Thrombosis among Carriers of Factor V Leiden
Further Information
Publication History
Received
14 April 2000
Accepted
28 April 2000
Publication Date:
11 December 2017 (online)
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References
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- 4 Hessner MJ, Dinauer DM, Luhm RA, Endres JL, Montgomery RR, Friedman KD. The prothrombin 20210GA genotype acts as a cooperative risk factor for venous thrombosis among factor V 1691GA (Leiden) carriers. Br JHaemat 1999; 106: 237-9.
- 5 Girard TJ, Eddy R, Wesselschmidt RL, MacPhail LA, Likert KM, Byers MG, Shows TB, Broze GJ. Structure of the human lipoprotein-associated coagulation inhibitor gene Intron/exon gene organization and localization of the gene to chromosome 2. J Biol Chem 1991; 266: 5036-41.
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- 7 Kleesiek K, Schmidt M, Getting C, Schwenz B, Lange S, Muller-Berghaus G, Brinkman T, Prohaska W. The 536 C→T transition in the human tissue factor pathway inhibitor (TFPI) gene is statistically associated with a higher risk for venous thrombosis. Thromb Haemost 1999; 82: 1-5.
- 8 Hessner MJ, Luhm RA, Pearson SL, Endean DJ, Friedman KD, Montgomery RR. Prevalence of prothrombin G202010A, factor V G1691A (Leiden), and methylenetetrahydrofolate reductase (MTHFR) C677T in seven different populations determined by multiplex allele-specific PCR. Thromb Haemost 1999; 81: 733-8.