Semin intervent Radiol 2007; 24(4): 433-436
DOI: 10.1055/s-2007-992332
MORBIDITY AND MORTALITY CASE

© Thieme Medical Publishers

Pulseless Electrical Activity Arrest after SVC Dilation

Brian Funaki1 , Taral Doshi1
  • 1Section of Vascular and Interventional Radiology, University of Chicago Hospitals, Chicago, Illinois
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Publikationsverlauf

Publikationsdatum:
11. Dezember 2007 (online)

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Superior vena cava syndrome (SVCS) is a constellation of symptoms that develops during stenosis or occlusion of the superior vena cava (SVC) or its main tributaries. Obstruction above the azygos vein causes a moderate increase in venous pressure because blood can be diverted through chest-wall veins into the thoracic and iliac veins and then enters the heart by way of the inferior vena cava and azygos system. Blockage below the level of the azygos vein is not well tolerated because a higher venous pressure is required to pump blood back to the heart through the only patent channel, the inferior vena cava.[1]

High central venous pressure produces a syndrome characterized by dyspnea, facial edema, venous distention of the chest wall and neck veins, and, less often, facial plethora, cough, arm edema, and central nervous system complaints.[2] [3] [4] [5] Although SVCS is a medical emergency that requires immediate diagnostic evaluation and therapy, it is rarely a life-threatening emergency. Symptoms are the most severe if the SVC is blocked quickly and venous collaterals do not have time to expand. Thus development of symptoms can be as rapid as a few weeks for a malignant tumor or gradual over several years for fibrosing mediastinitis.[1]

REFERENCES

Brian FunakiM.D. 

Section of Vascular and Interventional Radiology

University of Chicago Hospitals, 5840 S. Maryland Avenue, MC 2026, Chicago, IL 60637